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Volume 42 Issue 1
Jan.  2019
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Article Navigation >  Journal of Molecular Imaging  > 2019  >  42(1): 1-4
Xiaoyan WANG, Hua XIAO, Jiuhong GUO, Xiaofeng TAO. Application value of magnetic resonance imaging in patients with apical hypertrophy cardiomyopathy[J]. Journal of Molecular Imaging, 2019, 42(1): 1-4. doi: 10.12122/j.issn.1674-4500.2019.01.01
Citation: Xiaoyan WANG, Hua XIAO, Jiuhong GUO, Xiaofeng TAO. Application value of magnetic resonance imaging in patients with apical hypertrophy cardiomyopathy[J]. Journal of Molecular Imaging, 2019, 42(1): 1-4. doi: 10.12122/j.issn.1674-4500.2019.01.01
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Application value of magnetic resonance imaging in patients with apical hypertrophy cardiomyopathy

doi: 10.12122/j.issn.1674-4500.2019.01.01

  • Department of Radiology, Shanghai Ninth People's Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai 200011,China

  • Received Date: 2018-10-25
  • Publish Date: 2019-01-01
    Abstract
  • Objective To evaluate the clinical value of cardiac magnetic resonance (CMR) in the diagnosis of apical hypertrophic cardiomyopathy (AHCM). Methods Nine patients with AHCM from 2017 to 2018 in our hospital were successfully underwent magnetic resonance scanning. Myocardial thickness, ventricular atrial volume, left ventricular ejection fraction were measured. Myocardial involvement segments, the characteristics of myocardial first perfusion and delayed enhancement were evaluated. Results CMR showed apical hypertrophic in all the patients. These patients were divided into two groups: pure AHCH and mixed AHCH. Among them, 6 patients were pure AHCM, 3 patients were mixed AHCM. 3 patients had apical aneurysm. All patients underwent myocardial contrast enhancement scanning, 8 patients presented late gadolinium enhancement (LGE) which showed plaques and focal enhancement. 5 pure AHCH’s LGE were located in the apex. 3 mixed AHCH’s LGE were involved the central left ventricle and even the papillary muscles. Conclusions CMR is best diagnostic modality for AHCM. LGE can characterize areas of myocardial fibrosis, which plays an important role in the natural history of AHCM.

     

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